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FIGURE 2 Regarding improved mitochondrial activity, AML cells persistent to treatment with Ara-C in an NSG mouse model presented a higher rate and a genetic signature associated with OxPhos, accompanied by an increase in mitochondrial mass, membrane potential, and ROS production (Farge et al., 2017)

The peptide's antioxidant mechanism operates through activation of the nuclear factor erythroid 2-related factor 2 (Nrf2) pathway, a master regulator of cellular antioxidant responses

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