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Several reports revealed that synovial macrophage pyroptosis promotes OA inflammation in chondrocytes through cytokine secretion (e.g., IL-1, IL-18), EV release with bioactive molecules, and direct chondrocyte death induction, leading to cartilage damage and OA progression[39], [40], [41]

This proliferative phenotype was shown to be induced by ROS/RNS-dependent PI3K, ERK1/2, and AMPK activation 37

The leading causes of LREAs were ascites (43%), encephalopathy (22%) and variceal bleeding (11%)

Activation of PPARA-mediated autophagy reduces Alzheimer disease-like pathology and cognitive decline in a murine model

Highly efficient renaturation of beta-lactamase isolated from moderately halophilic bacteria