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849 CAT improves intestinal I/R injury by activating the transient receptor potential cationic channel subfamily V member 1 (TRPV1), enhancing GPX4 expression, and inhibiting ferroptosis

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The finding established that the GSH redox cycle is the primary intracellular antioxidant defence system in endothelial cells against peroxide-mediated oxidative stress, and remains a foundational reference for endothelial oxidative stress research using GSH manipulation

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