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Previously, we showed that the sensitivity of ARID1A-deficient cells to eprenetapopt (APR-246) is independent of TP53 status 15

In vivo proof of a powerful proinflammatory mechanism and a new anti-inflammatory strategy

Details of the ICD codes for intestinal obstruction are listed in Supplementary Table S2

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It is at the vanguard of prospective therapeutic treatments for treating metabolic diseases, obesity, and possibly even the cellular mechanisms involved in aging because of its ability to block the enzyme Nicotinamide N-methyltransferase (NNMT)