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We would like to acknowledge the Central Instrumentation Facility (CIF), Central Electrochemical Research Institute (CECRI) Karaikudi, for the TEM analysis

doi: 10.1586/14737175.8.6.957

In particular, we demonstrated that decrement in GSH level, induced pharmacologically by treatment with buthionine sulfoximine (BSO) or metabolically by fasting, resulted in increased NO bioavailability at the muscle level, a condition that could be detrimental [44]

A decline in GPX4 function directly increases the risk of ferroptosis, making its expression changes in OA a significant marker for assessing this risk

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