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The scientific justification for the use of a linear non-threshold extrapolation model stems from several sources: the similarity between carcinogenesis and mutagenesis as processes which both have DNA as target molecules, the strong evidence of the linearity of dose-response relationships for mutagenesis, the evidence for the linearity of the DNA binding of chemical carcinogens in the liver and skin, the evidence for the linearity in the dose-response relationship in the initiation stage of the mouse 2-stage tumorigenesis model, and the rough consistency with the linearity of the dose-response relationships for several epidemiological studies

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